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If you’ve ever witnessed someone suffer a stroke, you understand the humbling nature of this disease. It can reduce the mightiest human being to an immobile, helpless creature. Impairment of crucial functions like speech, walking, and control of bowel and bladder can wrench control from the body in a moment. Even perpetually youthful TV personality Dick Clark was struck down by stroke at age 75, despite the outward appearance of perfect health. Clark’s stroke resulted in a six-week hospital stay and, judging from fragmented reports, significant disability. Stroke can be like a devastating fire that strikes without warning, leaving only smoldering rubble. Stroke can so ravage basic bodily functions that often all you can hope for is to regain a portion through rehabilitation. The disease process that underlies stroke requires decades—30 or 40 years—to develop. With that much lead time, why aren’t we better able to detect or stop this crippling disease? The truth is that we are able to predict many, if not most, strokes. Advances in imaging technology allow detection of atherosclerotic plaque that cause stroke years before it becomes a threat. Progress in deciphering the causes of stroke has also leapt forward. Unfortunately, your neighborhood physician still focuses on diagnosing the crisis rather than anticipating it. Physicians prefer to deal with catastrophes and are just not that interested in prevention. Most physicians ask: “Is it time to operate or not?” The medical community obsesses over procedures like carotid endarterectomy (surgical removal of plaque) or carotid stents. Even when a person is afforded the warnings of a “mini-stroke”, or transient ischemic attack (TIA), little more is done once it’s determined that surgery is not necessary—even though this person has high risk for future stroke (50% over 10 years). Let’s flip-flop this approach to stroke. Procedures represent a failure of prevention! Where do strokes come from? Stroke develops when some portion of the brain is deprived of blood. This usually results from a tiny bit of debris that dislodges from an atherosclerotic plaque along the walls of an artery (the same sort that accumulates in coronaries causing heart attack). The sources of debris have been a subject of controversy, but new imaging technologies have settled the question. Any blood vessel that leads from the heart to the brain can be a source. The two carotid arteries on both sides of your neck are a frequent source, as these arteries are prone to develop plaque. (Our discussion will be confined to what are called thromboembolic, or ischemic, strokes, i.e, strokes that occur from plaque that fragments, sending debris to the brain, and will not include the far less common hemorrhagic strokes due to rupture of small vessels in the brain, nor will we discuss atrial fibrillation and other heart causes of stroke. The thromboembolic strokes we discuss cause around 88% of all strokes.) Over the last 10 years, the aorta has been recognized as another important source of stroke. The aorta is the main artery of the body whose branches go to the head, arms, and legs. Atherosclerotic plaque is a live tissue that, through poor diet, inactivity, high cholesterol, overweight, etc., grows and becomes progressively more unstable. At some point, plaque fragments. Little bits break away, traveling to the brain. Fractured plaque also exposes its deeper structures to flowing blood, triggering blood clot formation, which in turn can also fragment and go to the brain. Atherosclerotic plaque is a prerequisite for the most common causes of stroke. If the majority of strokes originate from plaque, why not measure plaque to determine if you’re at risk for stroke? How can we easily, safely, and accurately measure plaque in the carotid arteries and aorta? And if plaque can be measured, can it be shrunk or inactivated to reduce or eliminate risk for stroke? How can plaque be measured? Just 20 years ago, the only practical method of identifying plaque in the carotids or aorta was through angiography, requiring catheters inserted into the body to inject x-ray dye. Angiography was impractical as a screening measure. CT scanning and magnetic resonance imaging (MRI) are emerging as exciting methods of imaging both carotids and aorta. Unfortunately, most centers and physicians are much more focused on the diagnostic uses of these technologies for people who have already suffered stroke or other catastrophe, and application of these devices for preventive uses is still evolving. One exception is when aortic calcification or aortic enlargement is incidentally noted on the increasingly popular CT heart scans; this is an important finding that can signal presence of aortic plaque. The one test that is widely available and can be performed in just about any center is carotid ultrasound. It’s simple, painless, and precise. Two basic observations can be made: 1. Plaque detection—Atherosclerotic plaque can be clearly visualized. If plaque blocks more than 70% of the diameter of the vessel, or if there are “soft” (unstable) elements in plaque, then stroke risk may be high enough to justify surgery or stents. However, if there are plaques that are less severe, substantial risk for stroke may still be present that can be reduced with preventive measures. 2. Carotid intimal-medial thickness—This is a measure of the thickness of the lining of the carotid artery in areas not involved by plaque, but often precedes the development of mature plaque. Carotid intimal-medial thickness also provides an index of body-wide potential for atherosclerotic plaque that can place you at risk for stroke. The aorta, for instance, cannot be well imaged by surface ultrasound but can still be a source for stroke. Increased carotid intimal-medial thickness and carotid plaque are closely associated with likelihood of aortic plaque. The Rotterdam Study of 4000 participants demonstrated that if carotid intimal-medial thickness is greater than normal (1.0 mm), then you can be at risk for stroke (and heart attack), even if no carotid plaques are detected. Carotid ultrasound is the one test you should consider that provides the most information with least effort. Ultrasound is harmless, painless, and can be obtained just about anywhere. Even if your doctor disagrees with your request for a carotid ultrasound, an increasing number of mobile services are popping up nationwide that make this test available for around $100. One important point: many scanners and interpreters will only report whether plaque is present or not. While this is important information, you should request that the carotid-intimal medial thickness be made as well. Not all centers can make this simple measure (because of software requirements), but it doesn’t hurt to try. Any amount of carotid plaque is reason to follow a preventive program, even if the plaque is insufficient to justify surgery. Can plaque be reduced? Can we shrink plaque in carotid arteries and aorta and thereby reduce, perhaps eliminate, these sources of stroke? That question is gaining momentum as effective therapies become available that pack real punch for reducing plaque. Study after study has now documented that plaque can be reduced and, with it, risk for stroke. Reduction in plaque of 10–20% is possible within a year or two. Let’s consider the most potent influences on carotid and aortic plaque growth that need to be considered in a plaque-reducing program. (I assume that you are a non-smoker—if you are a smoker, you first need to concentrate on quitting.) Hypertension Considerable experience documents the power of blood pressure-lowering for prevention of stroke. The most recently updated guidelines, the JNC–VII, recommends a blood pressure of 407 mg/dl heightens stroke risk six-fold. C-reactive protein (CRP) This measure of inflammation is proving to be a useful marker for identifying people at risk for stroke, with increased risk beginning at a level of 0.5 mg/l. High CRP also predicts more rapidly growing carotid plaque. Homocysteine Homocysteine is an important marker of increased likelihood of both carotid and aortic plaque, as well as stroke. In 1997, the European Concerted Action Project reported more than a doubling of stroke when homocysteine levels exceeded 12 mol/l. As homocysteine increases to 20 μmol/l, risk for stroke and heart attack increases an amazing 10-fold over that at a level of 9 μmol/l. Asymmetric dimethylarginine (ADMA) ADMA is recently discovered amino acid whose blood levels can skyrocket up to 10-fold in the presence of hypertension, metabolic syndrome, diabetes, high cholesterol and triglycerides, obesity, and high homocysteine levels. ADMA blocks the action of the amino acid, l-arginine. This mimicry reduces the availability of nitric oxide, a powerful dilator and protector of arteries. ADMA levels in the top 10% predict a six-fold heightened risk for future stroke, and ADMA levels in people with strokes are double that in other people. A carotid ultrasound study in 116 subjects showed that higher blood levels of ADMA are associated with more severe carotid plaque. Because of ADMA’s shared role across a variety of abnormal conditions, correction or blocking the action of ADMA has been suggested as a unique therapeutic tool to reduce stroke risk. Cholesterol Data suggest that lowering cholesterol with statin cholesterol-lowering drugs slows carotid plaque growth and reduce stroke risk approximately 22%. An interesting study from the Cardiovascular Institute at Mt. Sinai School of Medicine in New York using the precise measuring ability of MRI of the carotids and thoracic aorta showed an impressive 20% regression of plaque area with simvastatin (Zocor®) taken for two years. Although guidelines for cholesterol treatment recommend reduction of LDL cholesterol to 100 mg/dl in high-risk persons, a report from the Walter Reed Army Medical Center in Washington, DC, showed that carotid plaque was more effectively reduced when LDL cholesterol of 70 mg/dl or lower was achieved with statin cholesterol drugs. Lower LDL cholesterol may, therefore, be better. Treatment Strategies to Reduce Carotid and Aortic Plaque The essential question: How do we reduce carotid and aortic plaque? If we make this the focus of our efforts, many pieces begin to fall into place. If you’ve had any measure of carotid or aortic plaque such as a carotid ultrasound or aortic calcification on a CT heart scan, you know that you’re at increased risk for stroke. You also have a baseline for future comparison to gauge whether your program is working or not. Because most people have not one but several causes of carotid and aortic plaque, there is no one single treatment that effectively eliminates risk for stroke. Instead, most people require a comprehensive program of healthy diet, exercise, supplements, and medication when indicated. Here, we focus on the nutritional supplements that can be critical components of your plaque-reduction program. Fish oil Fish oil is a cornerstone of your stroke prevention program. Epidemiological observations suggest a strong relationship of fish intake and reduction of stroke risk. Carotid ultrasound studies demonstrate less carotid plaque with greater intakes of fish. A cleverly designed University of Southampton study made the fascinating observation that fish oil transforms the structure of carotid plaque. 150 people with severe carotid plaque scheduled for carotid endarterectomy (surgical removal of the plaque) were given fish oil, sunflower oil, or no treatment over several months while waiting for their procedure. (Delays in the British health system permitted this unique design.) Plaque was removed at surgery and examined. Participants taking fish oil had reduced inflammation in plaque and thicker tissue covering the fatty core, markers of more stable plaque. Those taking sunflower oil or no treatment had unstable plaques with greater inflammation and thinner, less sturdy covering tissue. This suggests that fish oil stabilizes carotid plaque, making it less likely to rupture and fragment. A standard capsule of fish oil (containing 300 mg of EPA + DHA) contains the same amount of omega-3s as a 3 oz serving of cod or halibut; three capsules (900 mg DHA + EPA) contain the equivalent of a serving of farm-raised salmon. The dose that seems to provide greatest protection from stroke, lowers triglycerides (that form abnormal lipoproteins; see above), and reduces fibrinogen, is four capsules per day (1200 mg EPA + DHA). Coenzyme Q10 (CoQ10) Although there are no data specifically addressing whether CoQ10 reduces plaque, it is a marvelously effective way to reduce blood pressure, one of the crucial factors causing carotid and aortic plaque growth. A pooled analysis of eight studies showed that, on average, CoQ10 in daily doses of 50–200 mg reduced systolic blood pressure by 16 mm Hg, diastolic pressure by 10 mm Hg. Data suggest that CoQ10 can reverse abnormal heart muscle thickening (hypertrophy), another manifestation of high blood pressure, strongly suggesting that CoQ10 has benefits beyond just reducing pressure. Supplements to correct the metabolic syndrome Weight loss is, without question, the most immediate and direct path to correction of this dangerous pre-diabetic condition. A drop of even 10–20 lbs yields improvements across the board: increased sensitivity to insulin, increased HDL, and reductions in triglycerides, CRP, fibrinogen, small LDL particles, and blood pressure. Diet and exercise are fundamental components of an effort to lose weight; low carbohydrate or reduced glycemic index diets (e.g., South Beach or Mediterranean) rich in fibers are clearly effective. Several supplements can amplify weight-reduction efforts and be useful adjuncts to your lifestyle program. Among them: White bean extract White bean extract blocks intestinal absorption of carbohydrates by 66%. 1500 mg twice a day with meals yields, on average, 3–7 lbs of weight loss in the first month of use. The only side-effect is excessive gas, due to unabsorbed starches. Glucomannan This unique fiber taken prior to meals absorbs many times its weight in water and thereby fills your stomach. You consequently take in less food. Most people lose around four lbs per month using 1500 mg prior to each meal. Interestingly, glucomannan also blunts the rise in blood sugar after meals, an effect that, by itself, may lead to weight loss. Be sure to take with plenty of water. DHEA This adrenal hormone is key to maintaining physical stamina, mood, muscle mass in men, and libido in women. A recent randomized, placebo-controlled study at Washington University in 56 subjects showed a 13% decline in abdominal fat (fat that drives resistance to insulin) measured by MRI with 50 mg of DHEA per day at bedtime, along with improved sugar control and lower insulin levels. Pectin, beta-glucan Pectin is the soluble fiber in citrus rinds, green vegetables, and apples, also available as a supplement. Beta-glucan is the soluble fiber of oats and is also available as a supplement. Both are wonderful fibers that provide feelings of fullness, lower cholesterol, slow release of sugars, and can yield modest weight reduction. A USC study in 573 subjects using carotid ultrasound showed that greater intake of healthy fibers like pectin and beta-glucan is associated with less carotid plaque growth. Folic acid, vitamins B6 and B12 Dr. Daniel Hackam at the Stroke Prevention and Atherosclerosis Research Centre in Ontario conducted a study using carotid ultrasound in 101 participants treated with folic acid 2.5 mg, vitamin B6 25 mg, and B12 250 mcg per day. Treatment resulted in plaque reduction, especially when homocysteine levels exceeded 14μmol/l at the start, compared to untreated participants who experienced substantial plaque growth. An attempt to clarify the role of homocysteine treatment was made through a National Institute of Health-sponsored study of stroke prevention. 3680 participants with a prior history of stroke were enrolled and given either a “low-dose” (20 mcg folic acid, 0.2 mg B6, 6 mcg B12) or a “high-dose” (2.5 mg folic acid, 25 mg B6, 400 mcg B12) regimen. Although starting homocysteine levels showed a graded association with stroke risk (higher homocysteine levels predicted greater stroke risk), the treatment groups experienced, on average, only a 2 μmol drop in homocysteine levels and no reduction in stroke risk over two years. The study investigators as well as critics have suggested that the study failed due to an insufficient treatment period and that the doses were too low. (The doses we use in our plaque reduction program are folic acid 2.5–5.0 mg, B6 50–100 mg, B12 1000–2500 mcg.) L-arginine L-arginine can be used to overpower the adverse effects of ADMA. L-arginine is emerging as an important carotid plaque-reversing tool. Early reports in animals showed that l-arginine completely halted growth of aortic plaque, and did so more effectively than lovastatin (a cholesterol-lowering drug). In humans, L-arginine reduces blood pressure, abnormal constriction of carotid and coronary arteries, blocks entry of inflammatory cells into plaque, increases sensitivity to insulin, and heightens exercise capacity. Following coronary angioplasty or stent placement, l-arginine results in up to 36% reduction in plaque growth. The average American takes in 5400 mg of l-arginine through food every day. Supplementing with doses of 3000–12,000 mg per day has proven useful to correct many of these phenomena. (We use a dose of 6000 mg of l-arginine powder, twice a day on an empty stomach, dissolved in water, for our plaque regression program.) Does this result in a reduction of stroke risk? The emerging data suggest that l-arginine is likely to exert a powerful plaque-reducing and stroke-preventing benefit, but we await more clinical trial data. Conclusion Reducing stroke risk by reversing carotid and aortic plaque is becoming an everyday reality, with better tools becoming available. To know whether you’re at risk, the best and most available imaging tool is carotid ultrasound, aiming to identify intimal-medial thickness >1.0 mm, or carotid plaque. Any degree of calcification of the aorta, such as on a CT heart scan, is another useful measure of risk. Treatment to reduce risk is multi-faceted but is based on examining all your sources of risk, including metabolic syndrome, small LDL, lipoprotein(a), and C-reactive protein. Fish oil is the one absolutely crucial ingredient in any stroke prevention program. Other supplements can be used in a targeted fashion, depending on the causes identified for your carotid or aortic plaque. Ideally, repeat scanning of your carotids should be done sometime after your program has begun to assess whether you’ve successfully achieved reversal of plaque growth. penis enlargment stretcher penis enhancement pump penis enhancement result pro solution pills side effects compare penis enlargement pills compare penile enlargment pills vimax penis enlargement tip easy enlargment free penile surgery way

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Sexual desire is one of the most essential and indispensable part and the most beautiful of our life. It is really very hard to hold back the desire of having sex. But there are many times when conditions do not support man for having sexual intercourse. One such condition is when the person is suffering from the problem called ‘erectile dysfunction’. This quote of ‘18 till I die’ has become a target for almost all men above forty suffering from erectile dysfunction or ED. And why not so! This is the age when the hormones are on their edge all the time and there is no problem whatsoever in doing what you want whether it is getting an erection or having sex with someone. But as the age passes, things also start to show their weaknesses. This is the time when man comes into the trap of erectile dysfunction. This ends an epoch of romance, passion and above all intimacy in relationship of couples. Not only this, because of erectile dysfunction and impotency, marriages has been wrecked, couples have split and individuals shattered. Erectile dysfunction is the inability of a man to uphold or reach an erection needed to satisfy his partner’s sexual needs. It is visible in the men in their early fifties. Generally, it is caused by damage of nerves, arteries, smooth muscles, and fibrous tissues. High blood pressure, diabetes, heart disease, kidney or liver disease, other vascular conditions, cigarette smoking, and too much alcohol drinking, are also in charge for restricting sufficient blood-flow into the vessels of the penis, and causing ED. It is very important to treat ED on time because it may lead to low self-esteem, sexual failure anxiety, and depression, stress and guilt and relationship clashes. Most of the men throughout the world suffering from ED now rely on a drug called viagra. 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Viagra comes under the class of drug called phosphodiesterase inhibitors, it controls and blocks an enzyme called phosphodiesterase-5 (pde-5), and helps the smooth muscles in the penis to get relaxed and widen, and that permits additional blood to run to the penis ensuing in faster and easier erection. Scientifically speaking, viagra which is an oral therapy for ED, is the citrate salt of sildenafil, which is a selective inhibitor of cyclic guanosine monophosphate-specific phosphodiesterase type 5 (pde5). Sildenafil is the main component of viagra, which blocks the activity of an enzyme, called phosphodiesterase type 5 (pde5), which is active in multiple tissues and cells. The effects of pde5 inhibition include improved production of nitric oxide, which is linked with enhanced functioning of the heart and blood vessels. Its dosage may fluctuate from person to person depending on the seriousness of their problem, but the most recommended dose is 50mg, some user may need the dose of 100mg if they fail to get the desired result on 50 mg pill. After consuming this pill, it may take half an hour to begin its work and the effect of single dose of viagra may last up to 4 hours. It should be taken an hour before sexual activity. But being a prescription drug it is advised and requested to take this pill as per doctor’s prescription. Viagra is not any aphrodisiac that can be used by anybody and certainly not meant for women or children. Viagra does not cure impotency and does not provide prevention from sexually transmitted diseases including aids. It is a cure only for erectile dysfunction. 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Osteoporosis is an indicator of the aging process. As the calcium in our body structure leeches, our bones become brittle and weak. Using only a calcium supplement cannot remedy or stop this threatening and crippling disease because the body cannot absorb and make use of the calcium without the presence of Silica. Data suggests that, instead of affecting healing, supplemental mineral calcium, in contrast, speeds up the leeching away of bone calcium and thus accelerates the degenerative process of osteoporosis and similar diseases that affect the connective tissues in the human body. In the case of osteoporosis, Silica can alleviate the pain and may restore the body’s self repair process. Osteoporosis indicators attack women primarily after menopause, but the deterioration process starts much earlier in their younger days. Countless women are dying of fractures caused by osteoporosis compared with cancer of the breast, cervix and uterus combined. It is important to know that in osteoporosis, thinning of the bones takes place due to the insufficient manufacturing of the surrounding protein medium in which calcium salts first deposit. An insufficient amount of calcium in the bone matrix causes an enlargement of canals and spaces in the bones, giving the bones a porous, thinned look. The weakened bone develops into a fragile state and can be broken by a minor injury. The bones may even fracture from ordinary pressure or stress. For objectives of re-mineralization in damaged bones, it is advised that a sufficient Silica supplement be taken daily. Bones are composed of mainly phosphorus, magnesium and calcium, but they also include Silica. Silica deposits minerals into the bones, especially calcium. It hastens the healing of fractures and also diminishes scarring at the location of a fracture. A good deal of research evidence indicates that through a transmutation process, Silica is turned into calcium when it is needed. It is for that reason that some scientists mention Silica as a precursor to calcium. Even when calcium is insufficient, the body can turn Silica into calcium that the bones need. cheap vigrx pill does penis elargement work penile enlargement result free penis enhancement technique manual pnis enlargement exercise sex vigrx real penis enlargement forum magna rx natural penis elargement and lengthening

There was a time in history when there was no awareness of the prostate let alone diagnosis and treatment. Countless numbers of men throughout the ages have suffered and died as a result of this ignorance. Through the miracles of modern medicine, today prostate disease is well defined and is no longer necessarily considered a death sentence. The most effective methods for handling prostate disease are knowledge and prevention. In our article here we will help you understand prostate disease, various diagnostic tools and provide recommendations for treatment. WHAT IS THE PROSTATE? The first step toward understanding how the prostate affects your health is to learn what the prostate is and how it functions. It is surprising how little many men know about such an important part of their anatomy. The prostate is an important segment of the male reproductive system. It is a gland that is located in the lower abdominal cavity, just below the bladder, in front of the rectum and behind the pubic bone. It partially surrounds the urethra. The urethra is the channel that carries urine to the penis from the bladder and it runs right through the prostate. A healthy prostate is about the size of a walnut, weighs approximately 1 ounce and is shaped similar to a donut. There are “seminal vesicles” that are attached to the prostate. They produce a protein that mixes with prostatic fluid which forms semen. How this works is that tubes from the testicles carry sperm up to the prostate where sperm is mixed with the seminal vesicle and prostatic fluids. This fluid is ejaculated during orgasm through ejaculatory ducts that connect to the urethra. In addition, the prostate helps to control the flow of urine. Nearly every man will experience some type of prostate problem during their lifetime. Men who are over forty-five may experience an enlargement of the prostate. While this is not a problem in itself, it is uncomfortable and can be a forerunner to other more serious maladies which we will discuss this further. SYMPTOMS OF PROSTATE PROBLEMS Enlargement of the prostate is called benign prostate hypertrophy, or BPH. While this growth is usually considered a nuisance, if a man experiences problems with burning, or difficult urination at any time, the prudent course of action is consulting a urologist. Other symptoms may be: • A feeling of having to push out urine. • A sensation that the bladder is not emptying. • Increased urinating, especially at night. • An intermittent starting and stopping of the urinary stream After a diagnosis of BPH, many men will just continue to live with the symptoms and subsequent discomfort. It is not a life threatening condition, and there are treatments. In some cases surgery might be considered if the enlargement is significant. DIAGNOSTIC TESTING Prostate cancer can occur in any man but there are certain “risk groups.” Younger African American men appear to have twice the risk and fatalities of Caucasians. Sadly, many are diagnosed before they reach the age of 50. Another risk group is men who have a family history of prostate cancer, placing them in the same group who may contract the disease before the age of 50. The only method to determine whether you are at risk for prostate cancer is diagnostic testing. The earlier you are screened the higher your chances of survival. penis elargement program prosolution pennis enlargement pills best enlargement exercise penis penis enargement tool penis enlargment traction device penile enlargement exercise penis enlargement surgery cost penis enargement patch natural penis elargement and lengthening

Prostate is not a problem just for elderly male population. Affecting 1 in 8 men, prostate disorders are much more common than would be expected in middle-aged individuals. The good news is that using natural treatments this battle can be a successful one. The natural approach to prostate problems involves four steps. Improving the blood and energy flow to the prostate region. This flow is affected by things like low back problems, constipation, scar tissue and injury. It can be improved by massage, manipulation therapies, acupuncture and the appropriate dietary changes. Diet targeted at improving the health of the prostate. Soy contains natural substances which help detoxify the harmful Di-Hydro-Testosterone (DHT), thereby reducing its stimulation for cell multiplication. It's a reality that Japanese men, who eat a plenty soy diet, have very low incidences of prostate problems. Therefore, products containing a high amount of soy have been recommended as preventing prostate enlargement. Vegetables containing carotenes and red-orange fruits are also associated with low prostate cancer incidence. Finally, adequate fiber in the diet, as well as pumpkin and sunflower seeds seem to improve prostate symptoms. Diet high in vitamins and supplements helping in prostate function. Zinc may be cancer protective as is required to utilize carotenes. In conjunction with vitamin B6, zinc also regulates the enzyme which converts testosterone to the harmful DHT. Vitamin E helps preserve the fatty acids and they are formatted in the human body to the messenger hormone prostaglandins which control, among other, inflammation. Use of some herbs with beneficial effects on the prostate. Saw palmetto berries contain substances which inhibit the conversion of DOT from testosterone. By consequence, they prevent the DOT that is produced from acting on the prostate, and cools inflammation in the gland itself. Saw palmetto is effective only in extract form (tinctures, capsules) while a tea made from the berries has no action. Saw palmetto is used extensively in US. In France and elsewhere in Europe, Pygeum africanum has been shown to work, again, by limiting the conversion of DHT and by reducing prostates enlargement and inflammation. This herb also is a mild antibiotic, which may explain its good effect in prostatitis as well as BAH. Finally, there is a Swiss extract of Utica devoice flower pollen (Carillon) which has potent anti-inflammatory actions and is effective in prostatitis again by blocking DOT. This type of approach may not cause any harm and it can be used as a preventive solution, but it is not a substitute for the advice of a physician or other medical professional.